◎ 2013年英文論文発表
1. Satoh J, Tabunoki H.
Molecular network of ChIP-Seq-based vitamin D receptor target genes.
Multiple Sclerosis Journal 19: 1035-1045, 2013. Cover photo
2. Satoh J, Tabunoki H.
A comprehensive profile of ChIP-Seq-based STAT1 target genes suggests
the complexity of STAT1-mediated gene regulatory mechanisms.
Gene Regulation and Systems Biology 7: 41-56, 2013.
3. Satoh J, Tabunoki H, Ishida T, Saito Y, Arima K.
Ubiquilin-1 immunoreactivity is concentrated on Hirano bodies and
dystrophic neurites in Alzheimer's disease brains.
Neuropathology and Applied Neurobiology 2013, in press.
4. Satoh J, Tabunoki H, Ishida T, Saito Y, Konno H, Arima K.
Reactive astrocytes express the potassium channel Kir4.1 in active
multiple sclerosis lesions.
Clinical and Experimental Neuroimmunology 4:19-28, 2013.
5. Satoh J. Molecular network of ChIP-Seq-based NF-kappaB p65 target genes
involves diverse immune functions relevant to the immunopathogenesis
of multiple sclerosis.
Multiple Sclerosis and Related Disorders 3: 94-106, 2013.
6. Satoh J, Kawana N, Yamamoto Y, Ishida T, Saito Y, Arima K.
A survey of TREM2 antibodies reveals neuronal but not microglial staining
in formalin-fixed paraffin-embedded postmortem Alzheimer's brain tissues.
Alzheimer's Research and Therapy 5: 30, 2013.
7. Satoh J, Kawana N, Yamamoto Y.
Molecular network of ChIP-Seq-based EBNA1-target cellular geens supports
biological implications of EBV persistence in multiple sclerosis.
Clinical and Experimental Neuroimmunology 4: 181-192, 2013.
8. Satoh J. A possible role of microgliopathy in the pathogenesis of Nasu-Hakola
disease.
Clinical and Experimental Neuroimmunology 4 (Suppl 1): 17-26, 2013.
9. Kawana N, Yamamoto Y, Ishida T, Saito Y, Arima K, Satoh J.
Reactive astrocytes and perivascular macrophages express NLRP3 inflammasome
in active demyelinating lesions of multiple sclerosis and necrotic lesions
of
neuromyelitis optica and cerebral infarction.
Clinical and Experimental Neuroimmunology 4: 296-304, 2013.
10. Satoh J, Kawana N, Yamamoto Y.
Pathway analysis of ChIP-Seq-Based NRF1 target genes suggests a logical
hypothesis
of their involvement in the pathogenesis of neurodegenerative diseases.
Gene Regulation and Systems Biology 7: 139-152, 2013.